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Spontaneous mutation of the Dock2 gene in Irf5
-/-
mice complicates interpretation of type I interferon production and antibody responses
Whitney E. Purtha
, Melissa Swiecki
, Marco Colonna
, Michael S. Diamond
,
Deepta Bhattacharya
Research output
:
Contribution to journal
›
Article
›
peer-review
72
Scopus citations
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Dive into the research topics of 'Spontaneous mutation of the Dock2 gene in Irf5
-/-
mice complicates interpretation of type I interferon production and antibody responses'. Together they form a unique fingerprint.
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Medicine and Dentistry
Interferon Production
100%
Antibody Response
100%
Cytokinesis
100%
Factor V
100%
Interferon Antibody
100%
Spontaneous Mutation
100%
Plasmacytoid Dendritic Cell
33%
B Cell Development
33%
Disease
16%
Guanine
16%
Virus Infection
16%
Autoimmune Disease
16%
Transcription Factors
16%
Breed
16%
Frameshift Mutation
16%
Homozygosity
16%
Interferon Regulatory Factor 5
16%
Keyphrases
Dedicator of Cytokinesis
100%
DOCK2
16%
Factor V Gene
16%
Human Autoimmune Diseases
16%
Duplication mutation
16%
Immunology and Microbiology
Spontaneous Mutation
100%
Biochemistry, Genetics and Molecular Biology
IRF5
16%
Neuroscience
Frameshift Mutation
16%