Zinc is an essential trace element required for protein synthesis and prenatal growth and development. To assess the role of zinc, as a factor in the pathogenesis of fetal growth retardation in maternal protein deficiency we investigated with an in vivo technique, placental transport of 65Zn in control and protein deficient pregnant rats. Our results indicate that the placental transport of zinc is unaltered by maternal protein deficiency. Furthermore, total fetal body zinc concentration was similar in pups from control and protein deficient mothers.
|Original language||English (US)|
|Number of pages||4|
|Journal||Nutrition Reports International|
|State||Published - 1983|
ASJC Scopus subject areas
- Medicine (miscellaneous)
- Food Science