LIVER TRANSPLANTATION FOR WILSON'S DISEASE

Robert W. Beart; Charles W. Putnam; K. A. Porter; Thomas E. Starzl

doi:10.1016/S0140-6736(75)90077-X

LIVER TRANSPLANTATION FOR WILSON'S DISEASE

Robert W. Beart, Charles W. Putnam, K. A. Porter, Thomas E. Starzl

Research output: Contribution to journal › Letter › peer-review

Original language	English (US)
Pages (from-to)	176-177
Number of pages	2
Journal	The Lancet
Volume	306
Issue number	7926
DOIs	https://doi.org/10.1016/S0140-6736(75)90077-X
State	Published - Jul 26 1975
Externally published	Yes

ASJC Scopus subject areas

General Medicine

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10.1016/S0140-6736(75)90077-X

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@article{44d5e51319454ee9b29f953976518bcc,

title = "LIVER TRANSPLANTATION FOR WILSON'S DISEASE",

author = "Beart, {Robert W.} and Putnam, {Charles W.} and Porter, {K. A.} and Starzl, {Thomas E.}",

note = "Funding Information: The complete correction, in our second patient, of all the classical clinical manifestations and biochemical abnormalities of Wilson{\textquoteright}s disease has not been reported with other forms of therapy.3 3 These observations lend support to the contention that this genetic disorder is liver-based. With the provision of a normal liver, body-copper is eliminated over a period of years. This work was supported by research grants from the Veterans Administration; by grants AI-AM-08898 and AM-07772 of the National Institutes of Health; and by grants RR-00051 and RR-0069 from the General Clinical Research Centers Program Health.of the Division of Research Resources, National Institutes of",

year = "1975",

month = jul,

day = "26",

doi = "10.1016/S0140-6736(75)90077-X",

language = "English (US)",

volume = "306",

pages = "176--177",

journal = "The Lancet",

issn = "0140-6736",

publisher = "Elsevier B.V.",

number = "7926",

}

TY - JOUR

T1 - LIVER TRANSPLANTATION FOR WILSON'S DISEASE

AU - Beart, Robert W.

AU - Putnam, Charles W.

AU - Porter, K. A.

AU - Starzl, Thomas E.

N1 - Funding Information: The complete correction, in our second patient, of all the classical clinical manifestations and biochemical abnormalities of Wilson’s disease has not been reported with other forms of therapy.3 3 These observations lend support to the contention that this genetic disorder is liver-based. With the provision of a normal liver, body-copper is eliminated over a period of years. This work was supported by research grants from the Veterans Administration; by grants AI-AM-08898 and AM-07772 of the National Institutes of Health; and by grants RR-00051 and RR-0069 from the General Clinical Research Centers Program Health.of the Division of Research Resources, National Institutes of

PY - 1975/7/26

Y1 - 1975/7/26

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U2 - 10.1016/S0140-6736(75)90077-X

DO - 10.1016/S0140-6736(75)90077-X

M3 - Letter

C2 - 49762

AN - SCOPUS:0016862517

SN - 0140-6736

VL - 306

SP - 176

EP - 177

JO - The Lancet

JF - The Lancet

IS - 7926

ER -

LIVER TRANSPLANTATION FOR WILSON'S DISEASE

ASJC Scopus subject areas

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