Human ATP synthase beta is phosphorylated at multiple sites and shows abnormal phosphorylation at specific sites in insulin-resistant muscle

Keyphrases

• Phosphorylation 100%
• ATP Synthase 100%
• Insulin Resistance 100%
• Multiple Sites 100%
• Oxidative Phosphorylation (OXPHOS) 33%
• Mitochondrial Dysfunction 25%
• Type 2 Diabetes Mellitus (T2DM) 16%
• Skeletal muscle 16%
• Insulin 16%
• Phosphorylation Sites 16%
• Site-specific Phosphorylation 16%
• Human Skeletal muscle 16%
• Immunoblotting 8%
• Binding Region 8%
• Human Disease 8%
• β Subunit 8%
• Diabetes 8%
• Obesity 8%
• Glucose Disposal 8%
• Tandem Mass Spectrometry 8%
• Non-diabetic 8%
• Nucleotide Binding 8%
• Protein Abundance 8%
• Hypothesis-driven 8%
• Skeletal muscle Biopsy 8%
• Type 2 Diabetic 8%
• Hyperinsulinemic-euglycemic Clamp 8%
• Obese Men 8%
• Reversible Phosphorylation 8%
• Nanospray 8%
• Healthy Obese 8%

Biochemistry, Genetics and Molecular Biology

• ATP Synthase 100%
• Skeletal Muscle 41%
• Oxidative Phosphorylation 33%
• Tyrosine Phosphorylation 16%
• Insulin Resistance 16%
• Western Blot 8%
• Downregulation 8%
• Tandem Mass Spectrometry 8%
• Beta Chain 8%