Effects of antioxidants on tobacco smoke - associated cardiovascular damage

Ronald R. Watson; Julian Rodriquez; Yinhong Chen

Effects of antioxidants on tobacco smoke - associated cardiovascular damage

Ronald R. Watson
, Julian Rodriquez
, Yinhong Chen

Health Promotion Sciences

Research output: Chapter in Book/Report/Conference proceeding › Chapter

Abstract

Tobacco smoke exposure contributes significantly to cardiovascular damage through a number of pathways, including direct toxicity to the endothelium by free radicals, indirect effect of platelet aggregation, as well as increased catecholamine, homocysteine, and chronic oxidative changes occurring in LDL cholesterol. All ultimately disrupt myocardial structure and function for increased coronary heart disease.

Original language	English (US)
Title of host publication	Environmental Tobacco Smoke
Publisher	CRC Press
Pages	59-68
Number of pages	10
ISBN (Electronic)	9781420042245
ISBN (Print)	9780849303111
State	Published - Jan 1 2000

ASJC Scopus subject areas

General Medicine
General Environmental Science

Cite this

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abstract = "Tobacco smoke exposure contributes significantly to cardiovascular damage through a number of pathways, including direct toxicity to the endothelium by free radicals, indirect effect of platelet aggregation, as well as increased catecholamine, homocysteine, and chronic oxidative changes occurring in LDL cholesterol. All ultimately disrupt myocardial structure and function for increased coronary heart disease.",

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N2 - Tobacco smoke exposure contributes significantly to cardiovascular damage through a number of pathways, including direct toxicity to the endothelium by free radicals, indirect effect of platelet aggregation, as well as increased catecholamine, homocysteine, and chronic oxidative changes occurring in LDL cholesterol. All ultimately disrupt myocardial structure and function for increased coronary heart disease.

AB - Tobacco smoke exposure contributes significantly to cardiovascular damage through a number of pathways, including direct toxicity to the endothelium by free radicals, indirect effect of platelet aggregation, as well as increased catecholamine, homocysteine, and chronic oxidative changes occurring in LDL cholesterol. All ultimately disrupt myocardial structure and function for increased coronary heart disease.

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M3 - Chapter

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SP - 59

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BT - Environmental Tobacco Smoke

PB - CRC Press

ER -

Effects of antioxidants on tobacco smoke - associated cardiovascular damage

Abstract

ASJC Scopus subject areas

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