Dioxin may promote inflammation-related development of endometriosis

Kaylon L. Bruner-Tran; Grant R. Yeaman; Marta A. Crispens; Toshio M. Igarashi; Kevin G. Osteen

doi:10.1016/j.fertnstert.2008.02.102

Dioxin may promote inflammation-related development of endometriosis

Kaylon L. Bruner-Tran
, Grant R. Yeaman
, Marta A. Crispens
, Toshio M. Igarashi
, Kevin G. Osteen

Research output: Contribution to journal › Review article › peer-review

67 Scopus citations

Abstract

Laboratory and population-based studies suggest that exposure to environmental toxicants may be one of several triggers for the development of endometriosis. We discuss evidence that modulation of the endometrial endocrine-immune interface could mechanistically link toxicant exposure to the development of this disease.

Original language	English (US)
Pages (from-to)	1287-1298
Number of pages	12
Journal	Fertility and Sterility
Volume	89
Issue number	5 SUPPL.
DOIs	https://doi.org/10.1016/j.fertnstert.2008.02.102
State	Published - May 2008
Externally published	Yes

Keywords

dioxin
Endometriosis
inflammation
leukocytes
progesterone

ASJC Scopus subject areas

Reproductive Medicine
Obstetrics and Gynecology

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10.1016/j.fertnstert.2008.02.102

Cite this

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title = "Dioxin may promote inflammation-related development of endometriosis",

abstract = "Laboratory and population-based studies suggest that exposure to environmental toxicants may be one of several triggers for the development of endometriosis. We discuss evidence that modulation of the endometrial endocrine-immune interface could mechanistically link toxicant exposure to the development of this disease.",

keywords = "dioxin, Endometriosis, inflammation, leukocytes, progesterone",

author = "Bruner-Tran, \{Kaylon L.\} and Yeaman, \{Grant R.\} and Crispens, \{Marta A.\} and Igarashi, \{Toshio M.\} and Osteen, \{Kevin G.\}",

year = "2008",

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T1 - Dioxin may promote inflammation-related development of endometriosis

AU - Bruner-Tran, Kaylon L.

AU - Yeaman, Grant R.

AU - Crispens, Marta A.

AU - Igarashi, Toshio M.

AU - Osteen, Kevin G.

PY - 2008/5

Y1 - 2008/5

N2 - Laboratory and population-based studies suggest that exposure to environmental toxicants may be one of several triggers for the development of endometriosis. We discuss evidence that modulation of the endometrial endocrine-immune interface could mechanistically link toxicant exposure to the development of this disease.

AB - Laboratory and population-based studies suggest that exposure to environmental toxicants may be one of several triggers for the development of endometriosis. We discuss evidence that modulation of the endometrial endocrine-immune interface could mechanistically link toxicant exposure to the development of this disease.

KW - dioxin

KW - Endometriosis

KW - inflammation

KW - leukocytes

KW - progesterone

UR - https://www.scopus.com/pages/publications/43549095903

UR - https://www.scopus.com/pages/publications/43549095903#tab=citedBy

U2 - 10.1016/j.fertnstert.2008.02.102

DO - 10.1016/j.fertnstert.2008.02.102

M3 - Review article

C2 - 18394613

AN - SCOPUS:43549095903

SN - 0015-0282

VL - 89

SP - 1287

EP - 1298

JO - Fertility and Sterility

JF - Fertility and Sterility

IS - 5 SUPPL.

ER -

Dioxin may promote inflammation-related development of endometriosis

Abstract

Keywords

ASJC Scopus subject areas

Access to Document

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