Skip to main navigation
Skip to search
Skip to main content
University of Arizona Home
Search content at University of Arizona
Home
Profiles
Departments and Centers
Scholarly Works
Activities
Grants
Datasets
Prizes
Current understanding of conventional outflow dysfunction in glaucoma
W. Daniel Stamer
, Ted S. Acott
Research output
:
Contribution to journal
›
Review article
›
peer-review
300
Link opens in a new tab
Scopus citations
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'Current understanding of conventional outflow dysfunction in glaucoma'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Keyphrases
Glaucoma
100%
Conventional Outflow
100%
Intraocular Pressure
66%
Contractility
66%
Nitric Oxide
33%
Gene Expression
33%
Trabecular Meshwork
33%
Tight
33%
Extracellular Matrix
33%
Dynamic Pressure
33%
Enzyme Activity
33%
Therapeutic Potential
33%
Therapeutic Targeting
33%
Drug Class
33%
Primary Open-angle Glaucoma
33%
Protease Activity
33%
Pressure Sensitivity
33%
Mechanical Stimulation
33%
Bioactive Lipids
33%
Pore Formation
33%
Rho Kinase Inhibitor
33%
Chemical Signals
33%
Signaling Mediators
33%
Outflow Pathway
33%
Passageway
33%
Mechanical Signals
33%
Trabecular Outflow
33%
Ocular Hypertension
33%
Intraocular Pressure Fluctuation
33%
Outflow Resistance
33%
Local Signaling
33%
Extracellular Matrix Materials
33%
Conventional Outflow Pathway
33%
Matrix Expression
33%
Medicine and Dentistry
Intraocular Pressure
100%
Glaucoma
100%
Extracellular Matrix
66%
Muscle Contractility
66%
Gene Expression
33%
Cytokine
33%
Nitric Oxide
33%
Lifespan
33%
Mediator
33%
Rho Kinase Inhibitor
33%
Proteinase
33%
Enzyme Activity
33%
Mechanical Stimulation
33%
Channel Gating
33%
Trabecular Meshwork
33%
Ocular Hypertension
33%
Open Angle Glaucoma
33%
Pharmacology, Toxicology and Pharmaceutical Science
Nitric Oxide
100%
Proteinase
100%
Cytokine
100%
Rho Kinase Inhibitor
100%
Glaucoma
100%
Open Angle Glaucoma
100%
Intraocular Hypertension
100%