Arginase: Marker, effector, or candidate gene for asthma?

Donata Vercelli

doi:10.1172/JCI200318908

Arginase: Marker, effector, or candidate gene for asthma?

Research output: Contribution to journal › Article › peer-review

Abstract

Microarray analysis of the expression profiles of lung tissue in two murine models of asthma revealed high levels of arginase I and arginase II activity, in association with IL-4 and IL-13 overexpression (see the related article beginning on page 1863), suggesting that arginine pathways are critical in the pathogenesis of asthma.

Original language	English (US)
Pages (from-to)	1815-1817
Number of pages	3
Journal	Journal of Clinical Investigation
Volume	111
Issue number	12
DOIs	https://doi.org/10.1172/JCI200318908
State	Published - Jun 2003

ASJC Scopus subject areas

General Medicine

Access to Document

10.1172/JCI200318908

Cite this

@article{445aff72142f425da39e19d4975d8758,

title = "Arginase: Marker, effector, or candidate gene for asthma?",

abstract = "Microarray analysis of the expression profiles of lung tissue in two murine models of asthma revealed high levels of arginase I and arginase II activity, in association with IL-4 and IL-13 overexpression (see the related article beginning on page 1863), suggesting that arginine pathways are critical in the pathogenesis of asthma.",

author = "Donata Vercelli",

year = "2003",

month = jun,

doi = "10.1172/JCI200318908",

language = "English (US)",

volume = "111",

pages = "1815--1817",

journal = "Journal of Clinical Investigation",

issn = "0021-9738",

publisher = "American Society for Clinical Investigation",

number = "12",

}

TY - JOUR

T1 - Arginase

T2 - Marker, effector, or candidate gene for asthma?

AU - Vercelli, Donata

PY - 2003/6

Y1 - 2003/6

N2 - Microarray analysis of the expression profiles of lung tissue in two murine models of asthma revealed high levels of arginase I and arginase II activity, in association with IL-4 and IL-13 overexpression (see the related article beginning on page 1863), suggesting that arginine pathways are critical in the pathogenesis of asthma.

AB - Microarray analysis of the expression profiles of lung tissue in two murine models of asthma revealed high levels of arginase I and arginase II activity, in association with IL-4 and IL-13 overexpression (see the related article beginning on page 1863), suggesting that arginine pathways are critical in the pathogenesis of asthma.

UR - https://www.scopus.com/pages/publications/0042243559

UR - http://www.scopus.com/inward/citedby.url?scp=0042243559&partnerID=8YFLogxK

U2 - 10.1172/JCI200318908

DO - 10.1172/JCI200318908

M3 - Article

C2 - 12813015

SN - 0021-9738

VL - 111

SP - 1815

EP - 1817

JO - Journal of Clinical Investigation

JF - Journal of Clinical Investigation

IS - 12

ER -

Arginase: Marker, effector, or candidate gene for asthma?

Abstract

ASJC Scopus subject areas

Access to Document

Other files and links

Fingerprint

Cite this