Arginase: Marker, effector, or candidate gene for asthma?

Research output: Contribution to journalReview articlepeer-review

58 Scopus citations


Microarray analysis of the expression profiles of lung tissue in two murine models of asthma revealed high levels of arginase I and arginase II activity, in association with IL-4 and IL-13 overexpression (see the related article beginning on page 1863), suggesting that arginine pathways are critical in the pathogenesis of asthma.

Original languageEnglish (US)
Pages (from-to)1815-1817
Number of pages3
JournalJournal of Clinical Investigation
Issue number12
StatePublished - Jun 2003

ASJC Scopus subject areas

  • Medicine(all)


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