Activation of α7 nicotinic acetylcholine receptors protects astrocytes against oxidative stress-induced apoptosis: Implications for Parkinson's disease

Neuroscience

• Oxidative Stress 100%
• Parkinson's Disease 100%
• Astrocyte 100%
• Nicotinic Acetylcholine Receptor 100%
• Glial Cells 37%
• Hydrogen Peroxide 25%
• Neurotrophic Factor 25%
• Neurodegeneration 12%
• Striatum 12%
• Substantia Nigra Pars Compacta 12%
• Microglia 12%
• Mitochondrial Permeability Transition Pore 12%
• Neurodegenerative Disorder 12%
• Dopaminergic Neuron 12%
• Mitochondrial Membrane Potential 12%
• Bcl-2 12%
• Treatment of Parkinson's Disease 12%
• Caspase-9 12%

Pharmacology, Toxicology and Pharmaceutical Science

• Nicotinic Receptor 100%
• Parkinson's Disease 100%
• Disease 40%
• Hydrogen Peroxide 40%
• Neurotrophic Factor 40%
• Symptom 20%
• Inflammation 20%
• Dopamine Receptor Stimulating Agent 20%
• Nerve Degeneration 20%
• Systemic Administration 20%
• Mitochondrial Permeability Transition Pore 20%
• Caspase 9 20%
• 1,2,3,6 Tetrahydro 1 Methyl 4 Phenylpyridine 20%

Keyphrases

• Oxidative Stress-induced Apoptosis 100%
• Oligodendrocyte Apoptosis 80%
• Mitochondrial Membrane Potential Loss 20%